Dr. Anthony Chaffee continues his conversation with Dr. Ashley, delivering powerful insights about cholesterol, heart disease, and the carnivore diet's therapeutic potential. Listeners discover the shocking truth about America's first heart attack death in 1912 and how processed foods and seed oils - not meat - correlate with the rapid rise of cardiovascular disease. The episode exposes how cholesterol has been wrongly vilified despite being essential for hormone production, brain function, and cellular health, while revealing how the sugar industry funded fraudulent research to shift blame away from their products.
The discussion tackles common concerns about thyroid function on ketogenic diets, explaining why many people actually see improvements in conditions like Hashimoto's thyroiditis when eliminating plant toxins called goitrogens. Dr. Anthony Chaffee shares clinical experience with over 100 Hashimoto's patients whose antibodies normalized on a carnivore diet, demonstrating how autoimmune conditions often stem from dietary toxicity rather than permanent disease states. The conversation concludes with compelling evidence from traditional populations who achieved 120+ year lifespans consuming primarily meat and dairy.
Key Takeaways
Heart disease was virtually unknown before 1912, yet became America's #1 killer within 20 years despite higher meat consumption in the 1800s, pointing to processed foods and seed oils as the real culprits
Cholesterol serves essential functions including hormone production (testosterone, estrogen, cortisol), brain structure, and cell membrane formation - low cholesterol can cause neurological damage and hormone deficiencies
LDL cholesterol naturally increases during fasting and ketosis because these lipoproteins transport fat for energy, representing improved metabolic function rather than disease risk
Plant sterols in vegetable oils trick the body into stopping cholesterol production, leading to hormone disruption and vitamin D deficiency even with adequate sun exposure
Thyroid dysfunction often improves on carnivore diets by eliminating goitrogens (thyroid-disrupting plant compounds) while reducing the body's need for thyroid hormone due to improved metabolic efficiency
Hashimoto's antibodies can normalize within months to a year on strict carnivore diets, suggesting autoimmune thyroid conditions result from dietary toxicity rather than permanent immune dysfunction
Heart Disease History: No Heart Attacks Before 1912
Cholesterol and LDL: Why They Rise on Carnivore Diet
Cholesterol's Vital Role: Hormones, Brain Function, and Cell Membranes
Gallstones: Why Low Fat Diets Cause Them, Not Fat Itself
Statins and Brain Damage: Alzheimer's Connection to Cholesterol
Ancel Keys Fraud: Sugar Industry's Attack on Saturated Fat
Cultural Diet Myths: India's High Diabetes vs Hong Kong's Longevity
Thyroid Function and Hashimoto's: How Carnivore Diet Heals Autoimmune
This is an auto-generated transcript from YouTube and may contain errors or inaccuracies.
[Music] There wasn't a single heart attack proven on autopsy anywhere in America before 1912. The first death from heart attack in America proven on autopsy was 1912. 20 years later, it's the number one killer in America. How the hell did that happen? We're eating more meat in the 1800s than we were uh 1910s, 20s, and 30s when this first came on the market and then became the number one killer in America. So, you can't blame it on meat, but you can show a strong association with processed foods, seed oils, and sugars. >> Welcome to the Dr. Ashley Show. >> Welcome back to the Dr. Ashley Show. I'm Dr. Ashley, and today we are going to be continuing our conversation with Dr. Anthony Chaffy from a few weeks ago, who was sharing all of his insights about nutrition, chronic diseases, and the carnivore diet specifically. So, if you missed part one, you want to make sure that you check it out. During that episode, we discussed the role of toxin in plants, how many modern chronic diseases stem from our diets, and the incredible potential of eating in line with our own biology. Today, in part two, we're picking up right where we left off, diving even deeper into how the carnivore diet could be the key to reversing many health issues. We talk about hypothyroid and many other issues that so many of us deal with. So, let's jump right in as we continue our conversation with Dr. Anthony Jaffy. What about eating more fat with cholesterol? People are so concerned about cholesterol going up. Uh what do you say about that? And what about I you know I find that I don't know maybe it's 20% of the population following this type of diet they have an increase in LDL but their other numbers improve like HDL goes up total cholesterol goes down triglycerides go down but that darn LDL >> what do you say to that what is that lean mass hyper respponders or or something like that >> well it's also we've also seen this in fasting you know when people fast for a number of days you see the exact same pattern HDL goes up, LDL goes up, triglycerides go down. >> So that's a natural state. Your body's trying to do this on its own. And it's so it's not because we're eating more fat and we're not eating more cholesterol. In fact, studies have shown that dietary cholesterol plays almost no role in our serum cholesterol. Um, and that's that's been recognized by the American Heart Association and other sorts of things as well. And and it's not like they're, you know, big on the meat diet either, but it's uh it's something that that is is recognized now. But when you just don't eat anything, the exact same changes happen and your LDL will go up, your HDL will go up, triglycerides will come down. So that's what your body's trying to do. And in fact, when you eat carbohydrates, it stops your body from doing that. You're actually going to make more triglycerides. You're going to be um changing your metabolism. So instead of running on your fat stores, you're going to be running on your carbohydrates that you've just eaten and the and the glycogen stores that you made from them. And when you switch that metabolic state to where you're running on your fat stores, some people call it fasting state. I don't call that a fasting state. I think that's our primary metabolic state. That's a primary metabolic state of nearly every animal in the world because carnivores make up 70% of the animal species in the world. They eat fat and protein. They don't take down a bunch of carbs, so they're in ketosis. They've done studies. Lions and wolves are in ketosis. But then, uh, you know, uh, herbivores as well, like I mentioned, the ones that eat fiber, they don't they don't absorb the glucose from the fiber. They their bacteria break that down. They absorb fat and protein. So, cows are in ketosis. Gorillas are in ketosis. We should be in ketosis, too. That's the natural metabolic state. You eat food, you store it in your fat, and then you run on your fat. That's the natural uh way of things. That's that's your energy stores. So when you're running on your fat, you have to transport th that fat from your ad atapost tissue to your liver, turn into glucose, glycogen, and ketones. And how does it get there? Fat is not water soluble. So it has to be transported in special transporting molecules called HDL, LDL. And so those are transport molecules. And when you're running on fat, then you need more of those molecules. That's part of becoming fat adapted. Your body is getting better. This is why people fast longer or they're on on a ketogenic diet longer. They start getting better and better and better energy. Part of that is because they're ramping up their cholesterol production, their HDL and LDL production so that they can physically move energy better. And so now they have more energy available and they have and they feel better. So that's part of it. Um, a friend of mine, Dr. Nick Norwitz, bright kid, he's a PhD biochemistry from Oxford. He's just in his final year in medical school at Harvard. So, you know, he's he's no dummy. He's part of the lean mass hyperresponders studies with Dave Feldman. And he wanted to demonstrate this metabolic uh pattern that when you just don't eat carbohydrates, this is going to be the natural thing. And so, the converse of that is if you add in carbohydrates, well, you should see the opposite, right? And so, he did. So he did an Oreo cookie experiment on himself and he published this and he said, "Hey, this is what I'm doing." Compare it to statins. And so he uh just ate a sleeve of Oreo cookies every day, including, you know, on top of this was extra calories. So he's still eating his highfat ketogenic diet, nearly a carnivore diet, but not quite. And he just added in extra 12 Oreo cookies, which has fat and sugar and all that other stuff and a lot of calories. And so he did that for two weeks and his cholesterol dropped by something like 74%. Right? His LDL and then he had a wash out period. Let his cholesterol come back up to, you know, nearly 400 and then he went on statins for 6 weeks and he didn't even get down nearly as low. It only dropped it down like 34%. So that shows that uh what's going on there. You know, it's a metabolic state change. That's it. And cholesterol is really good for you. Your entire body is made out of cholesterol. Your entire body requires cholesterol. Your brain is largely made out of cholesterol. Your cells are all made out of cholesterol. There's a lipid billayer that make up the cell membrane. That lipid in that lipid billayer is cholesterol. Every sex steroid hormone that you have, estrogen, progesterrogens, testosterone, cortisol, minerals, gluccocorticoids, DHEA, pregnenolone, all these things, they are all made vitamin D. They're all derived from cholesterol. If you don't have enough cholesterol, you don't have enough hormones. If you are eating plant fats, they have plant steriles, which is the plant version of cholesterol. And they're close enough to cholesterol that that our body stops making cholesterol. I mean, that's how important cholesterol is. Our body makes it. And so, when you have plant steriles, your body thinks, "Oh, we have enough cholesterol. We don't need all this stuff." And so, you don't make enough of it. Your hormones drop. Your vitamin D drops. You're not able to produce it and stimulate it. You can get out in the sun if you want to, but if you don't have enough cholesterol, you're not going to be able to create enough vitamin D. Um, bile is made out of it. That's why they blame gallstones on people eating fat because, oh, there's all this cholesterol. So, you all this cholesterol from your diet and it's just trying to stuff it in and into your bile and you get these gallstones. Well, that's not true. It actually comes from not eating enough fat because that's typically who gets gallstones. It's the people that are avoiding fat. So the four Fs are for the more common demographic that we see gallstones are female 40s fertile fat. And so what is that? It's a woman, middle-aged woman who's had a couple kids, probably has some extra baby weight, wants to lose it. And as we all know, if you want to lose fat, you have to stop eating fat, right? >> Wrong. So they stop eating fat. They may lose some weight. They starve themselves. They feel miserable. And then they just go, "This is I'm not even losing all that much weight." and they go back to eating what they were before and all they get this pain in their right side and they go to the doctor and they go like you got gallstones and it's like but you know how do I get gallstones ah because you've been eating too much fat no it's because they haven't been eating fat because again bile is required to absorb fat and that is so important that you have five organs all working in concert just to absorb fat so you have the stomach starts breaking down the food the liver makes bile gallbladder stores it and concentrates it and that's very important because if you don't get a kill every single day and it's a week or two before between meals, you still need to absorb the same amount of fat. So it concentrates it up to 20 times what it comes out of your liver as normally physiologically. Some people probably more than that and that becomes important later. But then also your pancreas makes enzymes that break things down like lipase and then your small intestine absorbs it. You have all five of those things working together. It's very energy dependent and uh that's not going to happen if your body doesn't want fat because if it didn't want fat, it simply wouldn't absorb it. Doesn't have to. It's working really hard to go out of its way to absorb the fat. And so it's making that bile. It's storing that in the gallbladder and it's concentrating it. And if you're not eating fat and you're not eating fat and you just continue to do this, then it's getting more and more concentrated. And what happens to any hyperconentrated solution at rest? forms precipitate. It forms crystals as anybody who took high school chemistry can tell you. And so that's what bile sludge is. That's what gallstones are. Even if you had a very severe prediliction to develop stones. If you just ate enough fat every day to account for all of the bile that you're making, it's physically impossible to get gallstones. It's not possible because it won't be there. there won't be any bile to turn into stones in your gallbladder. So, it's it's from not eating fat, from not not from too much fat. So, cholesterol is used to make bile. It's used to make your hormones. It's used to make every cell in your body. It's used to make your brain a large portion of your brain is cholesterol. the mileination around your axons which allow your uh nerve cells to communicate faster so that your brain works so that your body works. That mileination is from saturated fat and cholesterol mostly cholesterol. Um so you know like babies you have kids so when they're they're newborn they're just little couple months old very uncoordinated can't stall sort of falling over trying to grab things and they're all wobbly and things like that. That's because their nerves aren't mileelinated yet. And so it's that mileination that actually gives them that coordination. So I'm giving a signal down to my hand. It's very responsive because it's nearly instantaneous from my brain to my fingers. Uh whereas in a baby when it's not mileelinated, it takes longer to get that signal there, right? And so when you don't have enough cholesterol or you don't have enough B12, which only exists in meat, you actually get demination of your nerves and your axons. You can actually get nerve damage. You can actually get brain damage. And so there are just a plethora of studies in the literature talking about how if you disrupt your normal cholesterol metabolism in your brain, get very serious neuro developmental and neurocognitive issues and diseases. And yet now we're going out of our way to bring about a disruption of our normal uh cholesterol metabolism in our brain and our body. You know, we take statins which cross the bloodb brain barrier and stop your brain from making cholesterol. They don't just take out excess cholesterol from your blood, which you're not in excess if your body's making it naturally. It stops your body and your brain from making it in the first place. And so, if you stop your brain from making cholesterol, you won't have enough cholesterol to build and maintain your brain. In fact, there are cases in the literature showing that people, adults who have Alzheimer's and are on statins were taken off the statins and six weeks later, wouldn't you know, they didn't have Alzheimer's. >> Wow. >> And then they were put back on the statins, six weeks later, they had Alzheimer's again. Isn't that unlucky? >> Right. And so, this is something that we really need to re-evaluate. The evidence against cholesterol and for cholesterol causing heart disease is zero. There has not been a single study of experimental design that has ever shown a cause and effect relationship between either cholesterol, total cholesterol, LDL cholesterol, apple, anything involved with cholesterol or saturated fat and cardiovascular disease. None. There have been at least five randomized control trials with thousands of patients that have actually shown the opposite. Three of them showed that there was no benefit by reducing saturated fat and reducing cholesterol. And two of them, the larger, more well-designed studies, one with nearly 10,000 patients actually showed that as you reduce saturated fat and reduce cholesterol and LDL cholesterol, more people died of heart attacks and strokes as a result. And that was done in the late60s from 68 to 73. It was called the Minnesota coronary experiment. And the reason that wasn't taught to any of us in medical school or nursing school or anything else is because they buried it for 40 years. And it wasn't until 2014 that Dr. Ramston from the NIH actually found whispers of this and found that this study had been done and contacted one of the lead researchers children who were around and they were doctors and they found their dad's old stuff and found all the data from this this study back in the 60s and 70s. Interestingly enough, one of the other lead researchers on and then they published in 2014 in Nature um top medical journ or top journal and one of the lead researchers back in the 60s and 70s happened to be the guy who uh famously publicized the cholesterol heart hypothesis which is Anel Keys. He got on the cover of Time magazine and he was notorious for trying to destroy the reputations and careers of anybody who said anything against his theory and um and he did you know this was there's a battle for decades if you go back into the literature and Jamama you know New England Journal of Medicine all these other top journals at the time you go in the 50s and 60s it is hotly contested there are papers this is based on no scientific evidence this is really weak evidence we shouldn't be accepting this and Um and then you have Anel Keys who was on the payroll from the sugar companies. That is a known fact that he was on the payroll from the sugar companies. He was pushing the other side and uh because there were researchers saying that hey we think sugar is causing heart disease which is a brand new disease. There wasn't a single heart attack proven on autopsy anywhere in America before 1912. The first death from heart attack in America proven on autopsy was 1912. 20 years later, it's a number one killer in America. How the hell did that happen? We're eating more meat in the 1800s than we were uh 1910s, 20s, and 30s when this first came on the market and then became the number one killer in America. So, you can't blame it on meat, but you can show a strong association with processed food, seed oils, and sugars. And so, Anel Keys was being paid by the processed food companies and sugar companies to basically put out counter op sort of uh work, which is what the the tobacco companies did for decades. And uh it's the same it's the same sort of tactic. You know, you try to just pump out garbage research and you just muddy the waters. Oh, we have 10 studies that say yes and 10 studies that say no. I guess we need more studies. And in the meantime, I keep getting to sell you poison. Um so Anel Keys was part of that Minnesota coronary experiment. Hey guys, just want to take a second to thank our sponsor at Carnivore Bar. I don't promote many products because honestly all you need to be healthy is to just eat meat. For those times that you're out hiking, road tripping, or stuck at work and you want nutritious snack that is just meat, fat, and salt if you want it, the Carnivore bar is a great option. So, I like this product not because it's just pure meat, but also because I want the carnivore market to thrive as well. And the more we support meatonly products, the more meatonly products there will be available in the mainstream. So, if this sounds like something you'd like to get behind, check it out using my discount code, Anthony, to get 10% off, which also applies to subscriptions, giving you 25% off total. All right, thanks guys. They buried it. It didn't have the result that they wanted. And one of the authors was actually interviewed before he died and said, "Hey, why did this not get published?" And he had a moment of honesty. He just said, "Well, we know there's nothing. Was there something wrong with the design? Was it a bad study?" Like, no, no, study was good. There's nothing wrong with it. It's just that we were just really disappointed with the results. You can't do that, right? Um, so Anel Keys was part of it. They ended up taking him off. They published an abstract back in the 80s, but they took him off of that, but he was one of the lead researchers on that. And then, surprise, surprise, they bury this thing. And then he keeps pushing for the rest of his career. Cholesterol is bad. Cholesterol is bad. lower cholesterol, you know, go on the Mediterranean diet, you know, eat a lot of plants and um and and sugar is fine. And they even said that don't eat fat. What do you replace it with? You replace it with sugar. It's healthy. It's fine. It's a it's empty calorie, but you need those calories. Calories are important. Get them from sugar is just a con. And so there is not a single shred of experimental evidence showing a cause and effect relationship between cholesterol and heart disease or saturated fat and and cholesterol or or saturated fat and heart disease. Here's another one. This is really important. Everyone vilifies saturated fat. It is not the problem. It's actually really beneficial. So journal American College of Cardiology and others have put massive meta analyses and systematic reviews and umbrella studies and umbrella studies. So if you have really good studies, really good randomized control trials, you make a meta analysis of that, that's really topshelf evidence like, wow, we've got 40 good quality randomized control trials. So not junk in, junk out, it's high quality. So you get high quality out and you're getting this amazing meta analysis. But then you have other people doing other metaanalyses of other top randomized control trials. And then you make a meta analysis of those meta analyses. That's called an umbrella review. So that is really the tip of the pyramid there. And so they have done umbrella reviews of highlevel studies, high level meta analyses and high level randomized control trials looking at the relationship between saturated fat and cardiovascular disease. And you know what they found? Absolutely no relationship whatsoever between increased saturated fat consumption and cardiovascular disease. In fact, they found an inverse relationship between saturated fat and strokes. So, people that ate more saturated fat had less strokes. Less saturated fat, more strokes. And so, the conclusion from these authors was like there should be no arbitrary upper limit set on the consumption of saturated fat. It is safe at every level tested. So, this is a con and it's a con that has cost billions of people their health and probably hundreds of millions to billions of people their life and died early and prematurely. And it is something that we desperately have to turn around. >> Hey there. Need extra support staying hydrated while shedding pounds and maintaining weight loss? Introducing Dr. Ashley's Electrolyte Plus, which I created just for this purpose. Packed with evidence-backed ingredients like vitamin D, C and zinc for immune support, B vitamins for energy, and a blend of fullsp spectrum electrolytes and minerals to hydrate, stabilize glucose levels, and curb cravings. Visit drashleywellness.com and use code Ashley10 for 10% off your first purchase or 20% off your first subscription order with free shipping. Plus, enjoy a 30-day money back guarantee. Back to the show. What do you say about cultures who don't eat a lot of fat and do you eat more carbs? Like people will say, "Well, Japanese people are really healthy." Or people in France who eat a lot of pasta, um, you know, their saturated fat intake is lower, >> maybe in Japan probably. What do you say about that? >> Well, you know, interestingly, France is actually very high saturated fat content. And so Anel Keys got famous because he did this something called the Seven Nation study and that's not an experimental study. That's observational. So it could only generate a hypothesis. It can't show a cause and effect relationship. And you took seven nations, you say, "Look, the more saturated fat and cholesterol, you have this >> upward sloping curve, this exponential growth curve showing that there's more heart disease and heart attacks, right?" Um, but he had complete data for 22 countries. One of them was France which actually broke that graph because they had very high saturated fat and very low cardiovascular disease. And so it's actually if you plot all those it's actually just a scattering across the board. Interestingly enough also um he was defending the sugar companies on the pay of the sugar companies but in his data just for those seven nations had the exact same correlation between sugar consumption and heart disease as he found with fat and cholesterol and heart disease for those seven nations. And then if you look at all the nations there's no correlation at all between fat, cholesterol and heart disease. But there is a strong correlation with sugar and heart disease. Um and so uh that's interesting too. So he's just a fraud all the way around. But yes, you know, you can be relatively more or less healthy. You know, if you're eating less junk food, you're eating less sugar, eating less high octane garbage, you are going to be relatively better. But does that mean that people in Japan who eat a clean traditional Japanese diet are as healthy as they possibly could be or they're just relatively better than people eating a junk food garbage diet? I think it's a relative improvement as opposed to an absolute improvement. >> Um but you know people in Okinawa, they said these are part of the blue zones. They say, "Oh, they're just eating a bunch of yams." No, actually they eat a ton of pork and fish. They actually have very high meat diet. >> And so um they actually do eat a lot of meat. Um but there are places like you know to use your example that eat more plant-based that eat far less saturated fat and much less meat in general like India you have an entire subcontinent of India is 1.4 4 billion people. So big population sample. And the interesting thing about them is they have a pretty similar diet. The standard Indian diet is vegetarian. >> Diabetes, don't they? >> They have the highest some of the highest diabetes rates in the world. It's on the highest metabolic syndrome uh rates in the world. They have over 33% of their population that has been tested. And a lot of people live rurally. They're very poor. They don't have access to medicine. 33% of the people tested had metabolic syndrome. And 25% in urban and rural areas had diabetes. Right? So in America, we're all fat, sick, McDonald's everywhere. All these terrible, horrible Americans, the worst of the worst. Wrong. Americans are 9% diabetic. India, plant-based India that only eats three kilos of meat per person per year on average. 25% diabetes rates. And the heart disease is out of control there. One in four people from India will die of a heart attack. One in three men from India will die of a heart attack. So if we want to look at plant-based versus meat, why don't we look at India? 1.4 billion examples. And then we can look at Hong Kong that eats the most meat per person per year and has the highest life expectancy on Earth. India has one of the lower ones. It's like 66 years. And Hong Kong is in the upper 70s, right? And they eat more meat. And there's in fact there was a study out of I think it was University of Adelaide in Australia a couple years ago that looked at 175 different countries they looked at all these different sorts of metrics and they found that in all 175 countries when corrected for socioeconomic sort of standards and all these other sorts of confounding factors that in every single country all 175 that the higher meat consumption equated directly with a an increased correlation in longevity and health. So, more meat equals longer life and better health. And we see traditional populations like the Messiah, you know, that you you talk to them and I' I've had friends do research trips out there and they would say when people die of old age, they're not getting killed by a lion or something. They don't die from heart disease and cancers and diabetes and things like that. Cancer is just really not known in those communities when living naturally. um they live to be 90, 100 or over 100 very readily and they're out active adults, you know, with their herds and migratorial. They have to keep moving all the time. So you have 100-year-old people walking with a pack on their back, right? Very healthy people into late age, but they have a a lower average life expectancy from birth because infant mortality rate is a lot higher when you're living out in the bush and you're dealing with lions and malaria and things like that. >> Yeah. But the age that people live to when they die of old age is far higher than what we're seeing in Western countries. And we saw that with the Native Americans as well. They were, you know, they had records of them living to be 110, 120, sometimes as old as 137 years old with Chief John Smith Whitewolf who was who died in the 1920s. And for decades they were saying how old he was and his family were saying how old he was. And people were like, "Wow, that's pretty old." And he just kept going and kept going and kept going. Um, and so people look back retrospectively and say, "Well, that's probably garbage because of X, Y, and Z." Well, you can say that, but there's no hard evidence one way or the other. We don't have a birth certificate on him because they didn't have birth certificates where he was living and when he was born. And uh, but we only have their records. And they say that he was, you know, 137. And also he describes in multiple different accounts in multiple different times his experiences fighting in the War of 1812. Right? So, if you're an adult and you fight in the War of 1812 and you die in the 1920s, you're pretty damn old. >> And it's it sounds far-fetched, >> but we now know as geneticists, what I was taught in genetics class 24 years ago >> is that humans actually are genetically designed to live 120 years on average based on the length of our tim. So, >> if they're just lying and they're just making things up, it's very interesting that they're all saying they just make it around that 110, 120, 130 year mark, which is an average 120. Um, Herododus, you go back in history, Herodus was the first historian from ancient Greece, and he chronicled an interaction between the Egyptian or sorry, the Persians and the Ethiopian uh king. So, diplomat from Persia came over and was talking to the Ethiopian king. The kings asked them, you know, what do your people eat and how long do your people normally live? And he described growing wheat and making bread and said that their people would live, you know, typically around 70 years, pretty similar to what we do now and pretty similar to what we eat now. And the Ethiopian king, you know, sort of laughed at him and said, "Well, no wonder you live such short lives if all you eat is dirt because we only eat boiled meat from our cattle and we only drink milk and water." And our people would live 120 years, sometimes much longer. And so there's that number again, 120. And why is that? Because that's actually how long we're supposed to live if we just stay out of our own way. So an average life expectancy genetically means that if you just stay out of your own way and don't mess up, you should make it to 120 without doing anything special. You don't have to do biohacking or this isn't cold plunges and, you know, red light therapy. You just just just live your life. >> As long as you don't mess up, you should make it to 120. So why are we dying in our 60s and 70s? Why are we saying someone who's 85 and they die from all these chronic diseases that wow they had a good life? They died 40 years early. And so I think that's an actual catastrophe. So that's again going back to the poisons that are in plants. Well, you can't say that spinach is poisonous. I eat spinach salad all the time. People smoke all the time, too. They drink all the time, too. They do heroin all the time, too. Doesn't kill you right away unless you have an acute, you know, overload. But over the days, over the months, over the years, over the decades, this poison builds up and breaks down your system and you get COPD, you get heart disease, you get cancer. And we know these are known consequences of poisoning yourself with cigarettes and alcohol. And yet, for some reason, we get these same diseases from eating bread and potato chips and and even Brussels sprouts. And yet, we wonder what the hell is going on. It couldn't possibly be. It's the same thing. You're damaging yourself slowly over time. And slow poison is still poison. And if you died half a century early, you were killed by poison. And that's all there is to it. >> Thanks, Dr. Chaffy. >> Welcome >> so much. I really appreciate everything. I have one more question, though, and I know I need to let you go, but I can't let you go before this one. >> That's fine. >> Okay. It can be a short answer, but a lot of people say that eating ketogenic low carb tanks your thyroid. >> I hear that all the time. What do you say to that? Just no. No. Yeah, it's a hard no. The thing is is that well, you Professor Steve Finny out of UC Davis, who's been studying ketogenic diets for decades, has actually shown that you don't need you simply don't need as much thyroid hormone when you're in a state of ketosis because your body's working better. Your metabolism is working better. And that's what thyroid uh your thyroid hormone has to do, plays in with your metabolism. Things just are running more efficiently. You just don't need as much. And that's fine, too. what I actually see in my clinical practice because so common now hypothyroidism and thyroid dysfunction so common now and so most patients who come into me fresh will have some sort of thyroid dysfunction. >> Yeah. >> And every single one when they go, you know, when I can convince them, which I actually have a pretty good um and and very motivated patient population, so they're very willing to hear me out and to try something. So most of them do try a ketogenic carnivore diet and they all improve and then you have to ask why why are people having thyroid dysfunction. Okay. Well, it could be low iodine, malnutrition, right? It could be some of these, you know, different toxins and there's a whole class of plant toxins called groitrogens which cause goers. Goer is is an enlarged thyroid because it doesn't work properly. And so you have these poisons that stuff up the functionality of your thyroid and make it not work as well and you get hypothyroidism. Or you keep bringing these plant toxins and lectins and your body doesn't like those and your body attacks them with antibodies and those antibodies get cross reaction with your thyroid. You call that Hashimoto's. So it depends on what's causing it. But again, it all comes down to toxicities and malnutrition. >> Toxicities, you have grogens is a known classification. You can Google it. It's fine. It has a Wikipedia page. I'm sure people can look up grogens and these things cause thyroid dysfunction and goiters. So, this is toxicity, right? And then not enough iodine, malnutrition, and um and then also the autoimmune side of things, which is also part of toxicity because you're getting an immune reaction to these toxins. So, it all comes down again to toxicity, malnutrition. You get rid of those toxins, you improve your nutrition, most problems go away. And so when I see people go on a highfat meat-based ketogenic diet, carnivore diet, I I push that for my patients, uh, their thyroid improves. Hashimoto's goes away. And I Hashimoto is very underdiagnosed. That's the thing. You need to find out why you have low thyroid. And most doctors traditionally trained won't test uh for Hashimoto's until your TSH goes above 10. But above two >> doubles your risk of developing thyroid cancer. So that's not a good place to be. So you want to be at 6 7 8 9. Ah, we'll just wait. What are you waiting for? There is a problem. Find out what it is. Stop it before it becomes a disaster. But they wait for the wheels to fall off before they do anything about it. >> So no, when the when the engine check engine light comes on, you start hearing a weird rattling noise in the engine. That's when you go, right? You don't just wait till the whole thing blows up, right? Which >> if you have pre-diabetes, you need to do something about it then and not wait until it becomes full-blown. >> That's it. Or even if you have perfectly normal blood sugar and you have insulin resistance >> because you have elevated insulin for 101 15 years before you ever get pre-diabetes, >> you need to do you need to do something then because that elevated insulin is now disrupting over 100 different mechanisms in your body. You need that insulin back down. And so you're getting all sorts of metabolic distress just from the insulin even before you get to the pre-diabetes. Then the pre-diabetes comes and the high heightened blood sugar directly damages your body. It's actually the blood sugar that kills diabetics, damages the body. The glucose molecules physically fuse to other molecules called glycation and it permanently damages them. It causes AES, advanced glycation end products. >> And so those build up and the body breaks down. And so this is why you lose your toes, you get your feet amputated, then your legs, then your kidneys start to fail, you go on diialysis, then you have to get kidney transplant, get heart disease, you get diabetic retinopathy, you go blind, Alzheimer's now being called type three diabetes because of insulin resistance of the brain. We see this on PET scans that there's reduced glucose uptake in the brain and people that are suffering from Alzheimer's. And then you die and it's this almost it's just a relief that someone dies after all of that. It all comes from just mildly elevated blood sugar. just that blood sugar is toxic to the body above a certain level. And so your body's desperately trying to keep it down below by ramping up your insulin. When you have a fire at your house, you know, you're not complaining to the fire department for spraying it with water and causing water damage. You're like, "Oh, just thank God the fire is out." That's what insulin's doing. And so you're just desperately trying to put out this fire so you don't die. The fire of high blood sugar. So you're getting damage from the insulin, but at least it's not burning the whole house down. But it does cause a lot of damage over time. If you keep restarting fires and keep getting water damage and fires and water damage and fires and water damage, like your house is going to be screwed very quickly. And that's what's happening to our bodies when we're when we're messing around with um blood sugar and insulin. Um but yeah, and so you don't wait for these things. I have, you know, just to to touch on the the thyroid thing, I have probably over a hundred patients with Hashimoto's specifically, and I can track their antibodies, and when they just go on a pure meat and water diet, especially like a ruminant animal diet like red meat and water, beef and lamb, and water, >> those antibodies just come right down. They can take months, it can take even over a year depending on how high their antibodies are, but as long as they're strict, those antibodies come right down to nothing. and then they become untraceable and it's like they don't have an autoimmune issue anymore. And if you're not making antibodies towards yourself, you really don't have it to speak of. It's like lead poisoning. You get the lead out. Well, you don't have lead poisoning anymore. Well, but what happens when you eat lead again? Yeah, you get lead poisoning again because it's not a disease. It's a toxicity. And when you reintroduce these things, your body makes antibodies towards them. >> Yeah, you're going to get a problem again. But that doesn't mean that it's always there and you can never cure it because it's not a cure situation. It's a toxin toxicity situation. So, you're removing all these toxins. You're improving your nutrition. You're going to improve your thyroid function there. You're removing causes of autoimmunity. Invariably, I see my patients, and I have hundreds of patients on a carnivore diet, their thyroid improves. I've never seen thyroid dysfunction on a carnivore diet. Hey guys, thank you very much for taking the time out to listen to what I had to say. If you like it, then please like and subscribe to my YouTube channel and podcast. And if you're on YouTube, then please hit that little bell and subscribe. And that'll let you know anytime I have a new video out, which should be every week, if not more. And if you could share this with your friends, that would help me get the word out and let me know that you like what I'm doing. Thanks again, guys.